Caffeine intake has been associated with a lower risk of several chronic metabolic diseases such as cardiovascular disease (stroke and ischemic heart disease), and type II diabetes.
The current study, which was carried out at Heinrich-Heine-University and the IUF-Leibniz Research Institute for Environmental Medicine in Duesseldorf, Germany, shows that caffeine, at a concentration equivalent to that in four cups of coffee, works to enhance the mitochondrial entry of a protein called p27.
P27 is protein that promotes effective mitochondrial function. It protects the cells of the heart and blood vessels against damage. The study was recently published in the open access journal PLOS Biology.